Inhibition of Autophagy Amplify Endoplasmic Reticulum Stress Dependent Cell Death in Cancer Cells
Prasun Guha and
Hafiz Ahmed
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Prasun Guha: Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine and Institute of Marine and Environmental Technology, USA
Hafiz Ahmed: Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine and Institute of Marine and Environmental Technology, USA
Biomedical Journal of Scientific & Technical Research, 2022, vol. 47, issue 4, 38819-38828
Abstract:
Tunicamycin mediated induction of endoplasmic reticulum (ER) stress promotes cell death of cancer cells. However, the apoptotic doses of tunicamycin shows immense toxicity to normal cells and thus limits its use as a cancer chemotherapeutic
Keywords: Journals on Medical Drug and Therapeutics; Journals on Emergency Medicine; Physical Medicine and Rehabilitation; Journals on Infectious Diseases Addiction Science and Clinical Pathology; Open Access Clinical and Medical Journal; Journals on Biomedical Science; List of Open Access Medical Journal; Journals on Biomedical Engineering; Open Access Medical Journal; Biomedical Science Articles; Journal of Scientific and Technical Research (search for similar items in EconPapers)
Date: 2022
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Persistent link: https://EconPapers.repec.org/RePEc:abf:journl:v:47:y:2022:i:4:p:38819-38828
DOI: 10.26717/BJSTR.2022.47.007545
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