Participation of No-Synthase in the Mechanisms of Oxidative Stress and Endothelial Injury in Rodent Peritonitis
Husakouskaya Ev,
N Ye Maksimovich and
Kavaliova Va
Additional contact information
Kavaliova Va: Department of Pathophysiology named after D. A. Maslakov, Grodno State Medical University, Belarus
Biomedical Journal of Scientific & Technical Research, 2024, vol. 54, issue 5, 46306-46310
Abstract:
The slow tendency of decrease in mortality from peritonitis may be due to an incomplete understanding of its pathogenesis. The participation of NO-synthase (NOS) in the mechanisms of development of oxidative stress and endothelial lesion during rodent peritonitis has not been fully studied, which may be of importance for researchers and for elaboration of adequate pathogenetic treatment of the pathology through the influence on the activity of this enzyme.
Keywords: Journals on Medical Drug and Therapeutics; Journals on Emergency Medicine; Physical Medicine and Rehabilitation; Journals on Infectious Diseases Addiction Science and Clinical Pathology; Open Access Clinical and Medical Journal; Journals on Biomedical Science; List of Open Access Medical Journal; Journals on Biomedical Engineering; Open Access Medical Journal; Biomedical Science Articles; Journal of Scientific and Technical Research (search for similar items in EconPapers)
Date: 2024
References: Add references at CitEc
Citations:
Downloads: (external link)
https://biomedres.us/pdfs/BJSTR.MS.ID.008603.pdf (application/pdf)
https://biomedres.us/fulltexts/BJSTR.MS.ID.008603.php (text/html)
Related works:
This item may be available elsewhere in EconPapers: Search for items with the same title.
Export reference: BibTeX
RIS (EndNote, ProCite, RefMan)
HTML/Text
Persistent link: https://EconPapers.repec.org/RePEc:abf:journl:v:54:y:2024:i:5:p:46306-46310
DOI: 10.26717/BJSTR.2024.54.008603
Access Statistics for this article
Biomedical Journal of Scientific & Technical Research is currently edited by Robert Thomas
More articles in Biomedical Journal of Scientific & Technical Research from Biomedical Research Network+, LLC
Bibliographic data for series maintained by Angela Roy ().