Routes of Neuroinflammation Autophagy and Calcium Dependent Mechanisms in Traumatic Brain Injury

Rajiv Kumar
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Rajiv Kumar: University of Delhi, New Delhi, India

Biomedical Journal of Scientific & Technical Research, 2024, vol. 54, issue 5, 46510-46512

Abstract: Neuroinflammation, immunosuppression, autophagy, and calcium-dependent mechanisms, are those cellular events, which occurred at the site of injury. Prolonged and delayed neuroinflammation promote macrophages and as a result, the activation of resident microglia cells comes into existence. The chain of these biological transformations enhances astrogliosis. The improper connected axonal and increased axonal transport proteins in the node originated prolonged swelling of injured axons and induced apoptotic cell death of neurons [1].

Keywords: Journals on Medical Drug and Therapeutics; Journals on Emergency Medicine; Physical Medicine and Rehabilitation; Journals on Infectious Diseases Addiction Science and Clinical Pathology; Open Access Clinical and Medical Journal; Journals on Biomedical Science; List of Open Access Medical Journal; Journals on Biomedical Engineering; Open Access Medical Journal; Biomedical Science Articles; Journal of Scientific and Technical Research (search for similar items in EconPapers)
Date: 2024
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Persistent link: https://EconPapers.repec.org/RePEc:abf:journl:v:54:y:2024:i:5:p:46510-46512

DOI: 10.26717/BJSTR.2024.54.008629

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