Ubiquinol and Autonomic Dysfunction in Hypertension and Heart Failure: A Short Review

Qiuhua Shen, Faith Rahman and Janet Pierce
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Janet Pierce: University of Kansas Medical Center, School of Nursing, USA

Open Access Journal of Neurology & Neurosurgery, 2020, vol. 13, issue 1, 1-2

Abstract: Coenzyme Q10 (CoQ10) is one of the most commonly used dietary and nutritional supplements and is known to be a potent antioxidant. There are two forms of CoQ10 including ubiquinone (oxidized form) and ubiquinol (reduced form) that are produced in the mitochondria [1-2]. CoQ10 is an essential component in the mitochondrial electron transport chain that plays a role in synthesis of Adenosine Triphosphate (ATP) and scavenging free radicals. A deficiency in CoQ10 can cause cellular energetic impairment and decreased antioxidant defensive mechanisms. Patients with heart failure with preserved ejection fraction (HFpEF) often have a reduction in CoQ10 and are prone to oxidative stress.

Keywords: Open Access Journal of Neurology; Journal of Neurology & Neurosurgery; Open Access Journal; juniper publishers; neurology open access journals; journal of neurology; neurology indexed journals; neurology scholarly journals; journal of neurology; neurology journals impact factor; juniper publishers reivew (search for similar items in EconPapers)
Date: 2020
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Persistent link: https://EconPapers.repec.org/RePEc:adp:joajnn:v:13:y:2020:i:1:p:1-2

DOI: 10.19080/OAJNN.2020.13.555851

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