Perinatal inflammation influences but does not arrest rapid immune development in preterm babies

S. Kamdar, R. Hutchinson, A. Laing, F. Stacey, K. Ansbro, M. R. Millar, K. Costeloe, W. G. Wade, P. Fleming and D. L. Gibbons ()
Additional contact information
S. Kamdar: King’s College London
R. Hutchinson: Homerton University Hospital
A. Laing: King’s College London
F. Stacey: Homerton University Hospital
K. Ansbro: Queen Mary University of London
M. R. Millar: Queen Mary University of London
K. Costeloe: Queen Mary University of London
W. G. Wade: King’s College London
P. Fleming: Homerton University Hospital
D. L. Gibbons: King’s College London

Nature Communications, 2020, vol. 11, issue 1, 1-14

Abstract: Abstract Infection and infection-related complications are important causes of death and morbidity following preterm birth. Despite this risk, there is limited understanding of the development of the immune system in those born prematurely, and of how this development is influenced by perinatal factors. Here we prospectively and longitudinally follow a cohort of babies born before 32 weeks of gestation. We demonstrate that preterm babies, including those born extremely prematurely (

Date: 2020
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DOI: 10.1038/s41467-020-14923-8

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