Dipeptidyl Peptidase-4, Heart Failure and Obesity- Current Status and Perspective

Carmine Finelli and Simone Dal Sasso
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Carmine Finelli: Department of Internal Medicine, Italy
Simone Dal Sasso: Independent Researcher, Italy

Biomedical Journal of Scientific & Technical Research, 2024, vol. 54, issue 5, 46439-46440

Abstract: Given that obesity increases the amount of blood that must be supplied to peripheral tissue, it is linked to a higher chronic cardiac workload. The increased demands of increased lean body mass are primarily responsible for the high cardiac output, which is sustained by an increase in ventricular mass, a high normal heart rate, and an increased stroke volume [1]. An increase in non-muscular tissue, which contributes to the development of electrical abnormalities, heart failure, and sudden death, is also implied by an increase in left ventricular (LV) mass [1]. One of the main risk factors for heart failure is obesity itself.

Keywords: Journals on Medical Drug and Therapeutics; Journals on Emergency Medicine; Physical Medicine and Rehabilitation; Journals on Infectious Diseases Addiction Science and Clinical Pathology; Open Access Clinical and Medical Journal; Journals on Biomedical Science; List of Open Access Medical Journal; Journals on Biomedical Engineering; Open Access Medical Journal; Biomedical Science Articles; Journal of Scientific and Technical Research (search for similar items in EconPapers)
Date: 2024
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Persistent link: https://EconPapers.repec.org/RePEc:abf:journl:v:54:y:2024:i:5:p:46439-46440

DOI: 10.26717/BJSTR.2024.54.008616

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