Neurodivergent Intelligence Framework: A Dimensional Framework for Understanding Cognition in Neurodivergent Profiles and Psychiatric Risk

Rochelle Bernard
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Rochelle Bernard: NeuroSync LLC

No hx72k_v1, SocArXiv from Center for Open Science

Abstract: Emerging evidence in neurodevelopmental research increasingly indicates that individuals diagnosed with Autism Spectrum Disorder (ASD) and Attention-Deficit/Hyperactivity Disorder (ADHD) may demonstrate amplified pattern recognition capabilities as a core cognitive feature. Historically, these capacities have been framed as deficits within prevailing diagnostic and clinical traditions rather than as expressions of differentiated cognitive architecture. However, the field has yet to develop a framework that accounts for the distinct types of capabilities across these profiles or the developmental conditions that shape their expression. The present paper introduces the Neurodivergent Intelligence Framework (NIF), proposing that individuals within these diagnostic categories exhibit two broad pattern recognition orientations: one externally oriented, functioning primarily through real-time environmental input, and the other internally oriented, operating through subconscious and reflective processing. Within each orientation, the domain toward which pattern recognition is directed produces distinct processing combinations, yielding four total combinations with different functional strengths, neurochemical dependencies, and vulnerability profiles. These differences are proposed to produce differential responses to early developmental environments, with long-term risk emerging when those environments fail to meet the cognitive demands of the individual’s specific architecture. Beginning in early developmental windows, exposure to social dynamics, parenting styles, and institutional contexts may shape neuroplasticity in ways that either support or suppress these cognitive profiles, with suppression potentially producing neurochemical consequences across dopamine, serotonin, oxytocin, and cortisol systems. When this suppression becomes chronic across critical developmental periods, the framework proposes that resulting cognitive and neurochemical strain may increase risk for impulsivity-driven coping responses and psychiatric comorbidity. In cases where these suppressive patterns coincide with trauma exposure and prolonged environmental misalignment, the framework further proposes elevated risk for conditions including schizophrenia, OCD, and bipolar disorder. The NIF also identifies elevated substance use disorder risk as a downstream consequence of chronic architectural misidentification and unmet neurochemical demands.

Date: 2026-06-04
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Persistent link: https://EconPapers.repec.org/RePEc:osf:socarx:hx72k_v1

DOI: 10.31219/osf.io/hx72k_v1

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