Mosaicism in Turner's syndrome

David Skuse and Patricia Jacobs
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David Skuse: Institute of Child Health, University College London Medical School
Patricia Jacobs: Institute of Child Health, University College London Medical School

Nature, 1997, vol. 390, issue 6660, 569-569

Abstract: Abstract Skuse and Jacobs reply — Henn and Zang argue that it is the presence of Y-chromosome sequences (presumably acting independently of sex steroids), rather than an imprinted gene on the X, that accounts both for male vulnerability to neurodevelopmental disorders of social cognition1 and for the poorer social adjustment of girls who have Turner's syndrome, with 45,Xm rather than 45,Xp. Unfortunately, without direct observations on the chromosome constitution of brain cells, Henn and Zang's hypothesis is not testable.

Date: 1997
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DOI: 10.1038/37516

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