Docking IκB kinases

Claus Scheidereit ()
Additional contact information
Claus Scheidereit: the Max-Delbrück-Centrum für Molekulare Medizin

Nature, 1998, vol. 395, issue 6699, 225-226

Abstract: The transcription factor NF-κB is activated in response to, among other things, stress. Normally, NF-κB is repressed by a protein called IκB which, in turn, is switched off by two protein kinases. Two studies now add another piece to the pathway, by showing that the interaction between all of these players could be facilitated by 'docking' proteins, which act as a kind of scaffolding.

Date: 1998
References: Add references at CitEc
Citations:

Downloads: (external link)
https://www.nature.com/articles/26121 Abstract (text/html)
Access to the full text of the articles in this series is restricted.

Related works:
This item may be available elsewhere in EconPapers: Search for items with the same title.

Export reference: BibTeX RIS (EndNote, ProCite, RefMan) HTML/Text

Persistent link: https://EconPapers.repec.org/RePEc:nat:nature:v:395:y:1998:i:6699:d:10.1038_26121

Ordering information: This journal article can be ordered from
https://www.nature.com/

DOI: 10.1038/26121

Access Statistics for this article

Nature is currently edited by Magdalena Skipper

More articles in Nature from Nature
Bibliographic data for series maintained by Sonal Shukla () and Springer Nature Abstracting and Indexing ().