p53 polymorphism and risk of cervical cancer

Alan Storey, Miranda Thomas, Ann Kalita, Catherine Harwood, Daniela Gardiol, Fiamma Mantovani, Judith Breuer, Irene M. Leigh, Greg Matlashewski and Lawrence Banks
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Alan Storey: Imperial Cancer Research Fund, Skin Tumour Laboratory
Miranda Thomas: International Centre for Genetic Engineering and Biotechnology
Ann Kalita: Institute of Parasitology and McGill Cancer Centre, McGill University
Catherine Harwood: Imperial Cancer Research Fund, Skin Tumour Laboratory
Daniela Gardiol: International Centre for Genetic Engineering and Biotechnology
Fiamma Mantovani: International Centre for Genetic Engineering and Biotechnology
Judith Breuer: St Bartholomew's and The Royal London Hospital School of Medicine and Dentistry, Queen Mary and Westfield College
Irene M. Leigh: International Centre for Genetic Engineering and Biotechnology
Greg Matlashewski: Institute of Parasitology and McGill Cancer Centre, McGill University
Lawrence Banks: International Centre for Genetic Engineering and Biotechnology

Nature, 1998, vol. 396, issue 6711, 532-532

Abstract: Abstract Storey et al. reply — These reports assess the frequency of the p53Arg allele in different populations and conclude that homozygous p53Arg is not a risk factor for cancer associated with human papilloma virus (HPV). The functional differences between the p53 isoforms that we have described1,2 provoked our initial epidemiological study. As we concluded then, it is crucial that investigations should be extended to different populations, and we are encouraged that such studies are underway.

Date: 1998
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DOI: 10.1038/25043

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