Mating, channels and kidney cysts
Scott W. Emmons () and
Stefan Somlo ()
Additional contact information
Scott W. Emmons: Albert Einstein College of Medicine
Stefan Somlo: Section of Nephrology, Yale University School of Medicine
Nature, 1999, vol. 401, issue 6751, 339-340
Abstract:
People with mutations in either of two proteins known as polycystin-1 and polycystin-2 develop polycystic kidney disease. Studies on the nematode worm and the frogXenopus laevisare now helping researchers to work out the normal functions of these proteins. It seems that polycystin-1-related proteins act as receptors, which regulate the activity of channels containing polycystin-2-related proteins.
Date: 1999
References: Add references at CitEc
Citations:
Downloads: (external link)
https://www.nature.com/articles/43810 Abstract (text/html)
Access to the full text of the articles in this series is restricted.
Related works:
This item may be available elsewhere in EconPapers: Search for items with the same title.
Export reference: BibTeX
RIS (EndNote, ProCite, RefMan)
HTML/Text
Persistent link: https://EconPapers.repec.org/RePEc:nat:nature:v:401:y:1999:i:6751:d:10.1038_43810
Ordering information: This journal article can be ordered from
https://www.nature.com/
DOI: 10.1038/43810
Access Statistics for this article
Nature is currently edited by Magdalena Skipper
More articles in Nature from Nature
Bibliographic data for series maintained by Sonal Shukla () and Springer Nature Abstracting and Indexing ().