Cell survival and a Gadd45-factor deficiency

Francesca Zazzeroni, Salvatore Papa, Enrico De Smaele and Guido Franzoso ()
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Francesca Zazzeroni: Ben May Institute for Cancer Research, and Gwen Knapp Center for Lupus and Immunology Research, University of Chicago
Salvatore Papa: Ben May Institute for Cancer Research, and Gwen Knapp Center for Lupus and Immunology Research, University of Chicago
Enrico De Smaele: Ben May Institute for Cancer Research, and Gwen Knapp Center for Lupus and Immunology Research, University of Chicago
Guido Franzoso: Ben May Institute for Cancer Research, and Gwen Knapp Center for Lupus and Immunology Research, University of Chicago

Nature, 2003, vol. 424, issue 6950, 742-742

Abstract: Abstract We and others have shown that the control of TNF-α-induced apoptosis by NF-κB/Rel transcription factors involves suppression of the JNK enzyme cascade1,2,3, and we have proposed that this suppression is mediated in part by Gadd45β/Myd118 (refs 1,4). Amanullah et al. suggest that the ablation of gadd45β has no effect on JNK activation and apoptosis by TNF-α and argue that the protective effects of NF-κB are mediated by factors other than Gadd45β.

Date: 2003
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DOI: 10.1038/424742a

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