An endocannabinoid mechanism for stress-induced analgesia

Andrea G. Hohmann (), Richard L. Suplita, Nathan M. Bolton, Mark H. Neely, Darren Fegley, Regina Mangieri, Jocelyn F. Krey, J. Michael Walker, Philip V. Holmes, Jonathon D. Crystal, Andrea Duranti, Andrea Tontini, Marco Mor, Giorgio Tarzia and Daniele Piomelli ()
Additional contact information
Andrea G. Hohmann: The University of Georgia
Richard L. Suplita: The University of Georgia
Nathan M. Bolton: The University of Georgia
Mark H. Neely: The University of Georgia
Darren Fegley: University of California
Regina Mangieri: University of California
Jocelyn F. Krey: Brown University
J. Michael Walker: Brown University
Philip V. Holmes: The University of Georgia
Jonathon D. Crystal: The University of Georgia
Andrea Duranti: University of Urbino Carlo Bo
Andrea Tontini: University of Urbino Carlo Bo
Marco Mor: University of Parma
Giorgio Tarzia: University of Urbino Carlo Bo
Daniele Piomelli: University of California

Nature, 2005, vol. 435, issue 7045, 1108-1112

Abstract: Stress cancelling Stress activates the neural systems that inhibit the sensation of pain, inducing natural analgesia. Some of these pathways involve natural opiates but now an endocannabinoid-based equivalent has been found. The enzymes that break up these natural marijuana-like compounds may be important therapeutic targets for stress-related disorders.

Date: 2005
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DOI: 10.1038/nature03658

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