TRIC channels are essential for Ca2+ handling in intracellular stores

Masayuki Yazawa, Christopher Ferrante, Jue Feng, Kazuhiro Mio, Toshihiko Ogura, Miao Zhang, Pei-Hui Lin, Zui Pan, Shinji Komazaki, Kazuhiro Kato, Miyuki Nishi, Xiaoli Zhao, Noah Weisleder, Chikara Sato, Jianjie Ma and Hiroshi Takeshima ()
Additional contact information
Masayuki Yazawa: Graduate School of Pharmaceutical Sciences, Kyoto University, Kyoto 606-8501, Japan
Christopher Ferrante: Robert Wood Johnson Medical School, New Jersey 08854, USA
Jue Feng: Graduate School of Medicine, Tohoku University, Miyagi 980-8575, Japan
Kazuhiro Mio: Neuroscience Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Ibaraki 305-8568, Japan
Toshihiko Ogura: Neuroscience Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Ibaraki 305-8568, Japan
Miao Zhang: Graduate School of Pharmaceutical Sciences, Kyoto University, Kyoto 606-8501, Japan
Pei-Hui Lin: Robert Wood Johnson Medical School, New Jersey 08854, USA
Zui Pan: Robert Wood Johnson Medical School, New Jersey 08854, USA
Shinji Komazaki: Saitama Medical University, Saitama 350-0495, Japan
Kazuhiro Kato: Graduate School of Medicine, Tohoku University, Miyagi 980-8575, Japan
Miyuki Nishi: Graduate School of Pharmaceutical Sciences, Kyoto University, Kyoto 606-8501, Japan
Xiaoli Zhao: Robert Wood Johnson Medical School, New Jersey 08854, USA
Noah Weisleder: Robert Wood Johnson Medical School, New Jersey 08854, USA
Chikara Sato: Neuroscience Research Institute, National Institute of Advanced Industrial Science and Technology (AIST), Ibaraki 305-8568, Japan
Jianjie Ma: Robert Wood Johnson Medical School, New Jersey 08854, USA
Hiroshi Takeshima: Graduate School of Pharmaceutical Sciences, Kyoto University, Kyoto 606-8501, Japan

Nature, 2007, vol. 448, issue 7149, 78-82

Abstract: Cell signalling requires efficient Ca2+ mobilization from intracellular stores through Ca2+ release channels, and it has been hypothesized that counter movement of ions across the sarcoplasmic reticulum neutralizes the transient negative potential generated by Ca2+ release. This paper identifies two trimeric intracellular cation channel subtypes that that could serve a critical role in such charge compensation.

Date: 2007
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DOI: 10.1038/nature05928

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