An elaborate pathway required for Ras-mediated epigenetic silencing

Claude Gazin, Narendra Wajapeyee, Stephane Gobeil, Ching-Man Virbasius and Michael R. Green ()
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Claude Gazin: Howard Hughes Medical Institute and Programs in Gene Function and Expression and Molecular Medicine, University of Massachusetts Medical School, 364 Plantation Street, Worcester, Massachusetts 01605, USA
Narendra Wajapeyee: Howard Hughes Medical Institute and Programs in Gene Function and Expression and Molecular Medicine, University of Massachusetts Medical School, 364 Plantation Street, Worcester, Massachusetts 01605, USA
Stephane Gobeil: Howard Hughes Medical Institute and Programs in Gene Function and Expression and Molecular Medicine, University of Massachusetts Medical School, 364 Plantation Street, Worcester, Massachusetts 01605, USA
Ching-Man Virbasius: Howard Hughes Medical Institute and Programs in Gene Function and Expression and Molecular Medicine, University of Massachusetts Medical School, 364 Plantation Street, Worcester, Massachusetts 01605, USA
Michael R. Green: Howard Hughes Medical Institute and Programs in Gene Function and Expression and Molecular Medicine, University of Massachusetts Medical School, 364 Plantation Street, Worcester, Massachusetts 01605, USA

Nature, 2007, vol. 449, issue 7165, 1073-1077

Abstract: A genome-wide RNAi screen identifies several genes required for Ras-mediated epigenetic silencing that encode cell signalling molecules, chromatin modifiers, transcriptional activators and repressors, and the DNA methyltransferase DNMT1.

Date: 2007
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DOI: 10.1038/nature06251

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