Toll-like receptor signalling in macrophages links the autophagy pathway to phagocytosis

Miguel A. Sanjuan, Christopher P. Dillon, Stephen W. G. Tait, Simon Moshiach, Frank Dorsey, Samuel Connell, Masaaki Komatsu, Keiji Tanaka, John L. Cleveland, Sebo Withoff and Douglas R. Green ()
Additional contact information
Miguel A. Sanjuan: Department of Immunology,
Christopher P. Dillon: Department of Immunology,
Stephen W. G. Tait: Department of Immunology,
Simon Moshiach: and
Frank Dorsey: St Jude Children’s Research Institute, Memphis, Tennessee 38105, USA
Samuel Connell: Department of Immunology,
Masaaki Komatsu: Laboratory of Frontier Science, Tokyo Metropolitan Institute of Medical Science, Bunkyo-ku, Tokyo 113-8613, Japan
Keiji Tanaka: Laboratory of Frontier Science, Tokyo Metropolitan Institute of Medical Science, Bunkyo-ku, Tokyo 113-8613, Japan
John L. Cleveland: The Scripps Research Institute, Jupiter, Florida 33458, USA
Sebo Withoff: Department of Immunology,
Douglas R. Green: Department of Immunology,

Nature, 2007, vol. 450, issue 7173, 1253-1257

Abstract: Autophagy contributes to host defence against infection. Toll like-receptor signals induce the recruitment of components of the autophagy pathway to phagosomes and facilitate phagosome lysosome fusion, resulting in killing of the invading organism.

Date: 2007
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DOI: 10.1038/nature06421

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