Schiff et al. reply

N. D. Schiff, J. T. Giacino, K. Kalmar, J. D.Victor, K. Baker, M. Gerber, B. Fritz, B. Eisenberg, T. Biondi, J. O’Connor, E. J. Kobylarz, S. Farris, A. Machado, C. McCagg, F. Plum, J. J. Fins () and A. R. Rezai
Additional contact information
N. D. Schiff: Weill Cornell Medical College
J. T. Giacino: JFK Johnson Rehabilitation Institute
K. Kalmar: JFK Johnson Rehabilitation Institute
J. D.Victor: Weill Cornell Medical College
K. Baker: Center for Neurologic Restoration, Cleveland Clinic Foundation
M. Gerber: JFK Johnson Rehabilitation Institute
B. Fritz: JFK Johnson Rehabilitation Institute
B. Eisenberg: JFK Johnson Rehabilitation Institute
T. Biondi: JFK Johnson Rehabilitation Institute
J. O’Connor: JFK Johnson Rehabilitation Institute
E. J. Kobylarz: Weill Cornell Medical College
S. Farris: Center for Neurologic Restoration, Cleveland Clinic Foundation
A. Machado: Center for Neurologic Restoration, Cleveland Clinic Foundation
C. McCagg: JFK Johnson Rehabilitation Institute
F. Plum: Weill Cornell Medical College
J. J. Fins: Weill Cornell Medical College
A. R. Rezai: Center for Neurologic Restoration, Cleveland Clinic Foundation

Nature, 2008, vol. 452, issue 7183, E1-E2

Abstract: Abstract Replying to: H. Staunton Nature 452, 10.1038/nature06574 (2008) Staunton1 highlights prior work applying deep-brain stimulation (DBS) in related thalamic and other subcortical structures in vegetative-state patients. We focused on patients who have plateaued at the upper end of the minimally conscious state at least one year after injury2, a group distinct from patients remaining in or just above vegetative state within the low end of the minimally conscious state. Patients remaining in a chronic vegetative state have anatomic pathology consistent with widespread neuronal death and cerebral disconnection3. In these patients, forebrain structures within the corticostriatopallidal–thalamocortical systems have been overwhelmingly damaged .

Date: 2008
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DOI: 10.1038/nature06575

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