Division and apoptosis of E2f-deficient retinal progenitors

Danian Chen, Marek Pacal, Pamela Wenzel, Paul S. Knoepfler, Gustavo Leone and Rod Bremner ()
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Danian Chen: Toronto Western Research Institute, University Health Network, and Laboratory Medicine and Pathobiology, University of Toronto
Marek Pacal: Toronto Western Research Institute, University Health Network, and Laboratory Medicine and Pathobiology, University of Toronto
Pamela Wenzel: Human Cancer Genetics Program, Immunology and Medical Genetics, Comprehensive Cancer Center, The Ohio State University, Columbus, Ohio 43210, USA
Paul S. Knoepfler: Davis, California 95616, USA
Gustavo Leone: Human Cancer Genetics Program, Immunology and Medical Genetics, Comprehensive Cancer Center, The Ohio State University, Columbus, Ohio 43210, USA
Rod Bremner: Toronto Western Research Institute, University Health Network, and Laboratory Medicine and Pathobiology, University of Toronto

Nature, 2009, vol. 462, issue 7275, 925-929

Abstract: E2f transcription factors The in vivo function of E2f transcription factors has been a matter of debate. They are generally thought to be essential for cell division, but in vivo experiments using the mouse retina as a model tissue now show, surprisingly, that cells can still divide in the absence of E2f1–3, due to functional redundancy with Myc.

Date: 2009
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DOI: 10.1038/nature08544

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