Stem-cell-triggered immunity through CLV3p–FLS2 signalling

Horim Lee (), Ok-Kyong Chah and Jen Sheen ()
Additional contact information
Horim Lee: Massachusetts General Hospital, Harvard Medical School
Ok-Kyong Chah: Massachusetts General Hospital, Harvard Medical School
Jen Sheen: Massachusetts General Hospital, Harvard Medical School

Nature, 2011, vol. 473, issue 7347, 376-379

Abstract: Development and immunity linked The plant stem cells in the shoot apical meristem provide a self-renewable reservoir for leaf, stem and flower organogenesis and are generally protected against stresses and disease. The molecular basis of this immunity has now been discovered. The CLV3 peptide (CLV3p) signal — which is secreted by stem cells in the Arabidopsis shoot apical meristem and which normally controls stem-cell homeostasis through CLV1 and CLV2 receptors — also triggers innate immune signalling through the bacterial flagellin peptide receptor kinase FLS2. The CLV3p–FLS2 signalling acts independently from the stem-cell signalling pathway. These findings suggest co-evolution in peptide–receptor signalling for development and immunity.

Date: 2011
References: Add references at CitEc
Citations:

Downloads: (external link)
https://www.nature.com/articles/nature09958 Abstract (text/html)
Access to the full text of the articles in this series is restricted.

Related works:
This item may be available elsewhere in EconPapers: Search for items with the same title.

Export reference: BibTeX RIS (EndNote, ProCite, RefMan) HTML/Text

Persistent link: https://EconPapers.repec.org/RePEc:nat:nature:v:473:y:2011:i:7347:d:10.1038_nature09958

Ordering information: This journal article can be ordered from
https://www.nature.com/

DOI: 10.1038/nature09958

Access Statistics for this article

Nature is currently edited by Magdalena Skipper

More articles in Nature from Nature
Bibliographic data for series maintained by Sonal Shukla () and Springer Nature Abstracting and Indexing ().