The BAH domain of ORC1 links H4K20me2 to DNA replication licensing and Meier–Gorlin syndrome

Alex J. Kuo, Jikui Song, Peggie Cheung, Satoko Ishibe-Murakami, Sayumi Yamazoe, James K. Chen, Dinshaw J. Patel () and Or Gozani ()
Additional contact information
Alex J. Kuo: Stanford University
Jikui Song: Structural Biology Program, Memorial Sloan-Kettering Cancer Center
Peggie Cheung: Stanford University
Satoko Ishibe-Murakami: Structural Biology Program, Memorial Sloan-Kettering Cancer Center
Sayumi Yamazoe: Stanford School of Medicine
James K. Chen: Stanford School of Medicine
Dinshaw J. Patel: Structural Biology Program, Memorial Sloan-Kettering Cancer Center
Or Gozani: Stanford University

Nature, 2012, vol. 484, issue 7392, 115-119

Abstract: The ORC1 BAH domain is shown to be a module that recognizes a histone modification associated with replication origins, providing insight into the aetiology of Meier–Gorlin syndrome.

Date: 2012
References: Add references at CitEc
Citations: View citations in EconPapers (5)

Downloads: (external link)
https://www.nature.com/articles/nature10956 Abstract (text/html)
Access to the full text of the articles in this series is restricted.

Related works:
This item may be available elsewhere in EconPapers: Search for items with the same title.

Export reference: BibTeX RIS (EndNote, ProCite, RefMan) HTML/Text

Persistent link: https://EconPapers.repec.org/RePEc:nat:nature:v:484:y:2012:i:7392:d:10.1038_nature10956

Ordering information: This journal article can be ordered from
https://www.nature.com/

DOI: 10.1038/nature10956

Access Statistics for this article

Nature is currently edited by Magdalena Skipper

More articles in Nature from Nature
Bibliographic data for series maintained by Sonal Shukla () and Springer Nature Abstracting and Indexing ().