NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells

Stephanie C. Eisenbarth (), Adam Williams, Oscar R. Colegio, Hailong Meng, Till Strowig, Anthony Rongvaux, Jorge Henao-Mejia, Christoph A. Thaiss, Sophie Joly, David G. Gonzalez, Lan Xu, Lauren A. Zenewicz, Ann M. Haberman, Eran Elinav, Steven H. Kleinstein, Fayyaz S. Sutterwala and Richard A. Flavell ()
Additional contact information
Stephanie C. Eisenbarth: Yale University School of Medicine
Adam Williams: Yale University School of Medicine
Oscar R. Colegio: Yale University School of Medicine
Hailong Meng: Yale University School of Medicine
Till Strowig: Yale University School of Medicine
Anthony Rongvaux: Yale University School of Medicine
Jorge Henao-Mejia: Yale University School of Medicine
Christoph A. Thaiss: Yale University School of Medicine
Sophie Joly: Inflammation Program, University of Iowa
David G. Gonzalez: Yale University School of Medicine
Lan Xu: Yale University School of Medicine
Lauren A. Zenewicz: Yale University School of Medicine
Ann M. Haberman: Yale University School of Medicine
Eran Elinav: Yale University School of Medicine
Steven H. Kleinstein: Yale University School of Medicine
Fayyaz S. Sutterwala: Inflammation Program, University of Iowa
Richard A. Flavell: Yale University School of Medicine

Nature, 2012, vol. 484, issue 7395, 510-513

Abstract: Dendritic cells in mice lacking NLRP10 are defective in exiting inflamed tissue, leading to impaired adaptive immunity; this could have a profound impact on the approach to treating allergic and autoimmune diseases.

Date: 2012
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DOI: 10.1038/nature11012

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