Inositol-1,4,5-trisphosphate receptor regulates hepatic gluconeogenesis in fasting and diabetes

Yiguo Wang, Gang Li, Jason Goode, Jose C. Paz, Kunfu Ouyang, Robert Screaton, Wolfgang H. Fischer, Ju Chen, Ira Tabas and Marc Montminy ()
Additional contact information
Yiguo Wang: Clayton Foundation Laboratories for Peptide Biology, The Salk Institute for Biological Studies, 10010 North Torrey Pines Road, La Jolla, California 92037, USA
Gang Li: Columbia University
Jason Goode: Clayton Foundation Laboratories for Peptide Biology, The Salk Institute for Biological Studies, 10010 North Torrey Pines Road, La Jolla, California 92037, USA
Jose C. Paz: Clayton Foundation Laboratories for Peptide Biology, The Salk Institute for Biological Studies, 10010 North Torrey Pines Road, La Jolla, California 92037, USA
Kunfu Ouyang: University of California San Diego, 9500 Gilman Drive, La Jolla, California 92093, USA
Robert Screaton: Children’s Hospital of Eastern Ontario Research Institute, University of Ottawa, Ottawa, Ontario K1H 8L1, Canada
Wolfgang H. Fischer: Clayton Foundation Laboratories for Peptide Biology, The Salk Institute for Biological Studies, 10010 North Torrey Pines Road, La Jolla, California 92037, USA
Ju Chen: University of California San Diego, 9500 Gilman Drive, La Jolla, California 92093, USA
Ira Tabas: Columbia University
Marc Montminy: Clayton Foundation Laboratories for Peptide Biology, The Salk Institute for Biological Studies, 10010 North Torrey Pines Road, La Jolla, California 92037, USA

Nature, 2012, vol. 485, issue 7396, 128-132

Abstract: Interactions between cyclic AMP and calcium signalling pathways mediated by the inositol 1,4,5-trisphosphate receptor are shown to regulate hepatic gluconeogenesis in fasting and diabetes.

Date: 2012
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DOI: 10.1038/nature10988

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