The yeast Fun30 and human SMARCAD1 chromatin remodellers promote DNA end resection

Thomas Costelloe, Raphaël Louge, Nozomi Tomimatsu, Bipasha Mukherjee, Emmanuelle Martini, Basheer Khadaroo, Kenny Dubois, Wouter W. Wiegant, Agnès Thierry, Sandeep Burma, Haico van Attikum () and Bertrand Llorente ()
Additional contact information
Thomas Costelloe: Leiden University Medical Center, Einthovenweg 20, 2333 ZC Leiden, The Netherlands
Raphaël Louge: Centre National de la Recherche Scientifique, Unité Mixte de Recherche 7258
Nozomi Tomimatsu: University of Texas Southwestern Medical Center, 2201 Inwood Road, NC7.214E, Dallas, Texas 75390, USA
Bipasha Mukherjee: University of Texas Southwestern Medical Center, 2201 Inwood Road, NC7.214E, Dallas, Texas 75390, USA
Emmanuelle Martini: CEA-DSV-Institut de Radiobiologie Cellulaire et Moléculaire
Basheer Khadaroo: Centre National de la Recherche Scientifique, Unité Mixte de Recherche 7258
Kenny Dubois: Centre National de la Recherche Scientifique, Unité Mixte de Recherche 7258
Wouter W. Wiegant: Leiden University Medical Center, Einthovenweg 20, 2333 ZC Leiden, The Netherlands
Agnès Thierry: Institut Pasteur, Unité de Génétique Moléculaire des Levures, Centre National de la Recherche Scientifique and University Pierre and Marie Curie-Paris, 25 rue du Docteur Roux, F75724 Paris Cedex 15, France
Sandeep Burma: University of Texas Southwestern Medical Center, 2201 Inwood Road, NC7.214E, Dallas, Texas 75390, USA
Haico van Attikum: Leiden University Medical Center, Einthovenweg 20, 2333 ZC Leiden, The Netherlands
Bertrand Llorente: Centre National de la Recherche Scientifique, Unité Mixte de Recherche 7258

Nature, 2012, vol. 489, issue 7417, 581-584

Abstract: Fun30 and SMARCAD1 are identified as chromatin remodellers that promote DNA end resection during DNA repair and preserve genome stability in yeast and humans, respectively.

Date: 2012
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DOI: 10.1038/nature11353

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