The Fun30 nucleosome remodeller promotes resection of DNA double-strand break ends

Xuefeng Chen, Dandan Cui, Alma Papusha, Xiaotian Zhang, Chia-Dwo Chu, Jiangwu Tang, Kaifu Chen, Xuewen Pan () and Grzegorz Ira ()
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Xuefeng Chen: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Dandan Cui: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Alma Papusha: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Xiaotian Zhang: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Chia-Dwo Chu: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Jiangwu Tang: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Kaifu Chen: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Xuewen Pan: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA
Grzegorz Ira: Baylor College of Medicine, One Baylor Plaza, Houston, Texas 77030, USA

Nature, 2012, vol. 489, issue 7417, 576-580

Abstract: Nucleolytic degradation of 5′ strands at DNA double-strand breaks in yeast is shown to be facilitated by the nucleosome remodeller Fun30, particularly within chromatin bound by the checkpoint adaptor protein known to inhibit resection, Rad9.

Date: 2012
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DOI: 10.1038/nature11355

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