Caspase-11 increases susceptibility to Salmonella infection in the absence of caspase-1

Petr Broz, Thomas Ruby, Kamila Belhocine, Donna M. Bouley, Nobuhiko Kayagaki, Vishva M. Dixit and Denise M. Monack ()
Additional contact information
Petr Broz: Stanford School of Medicine, Stanford University
Thomas Ruby: Stanford School of Medicine, Stanford University
Kamila Belhocine: Stanford School of Medicine, Stanford University
Donna M. Bouley: Stanford University
Nobuhiko Kayagaki: Genentech Inc.
Vishva M. Dixit: Genentech Inc.
Denise M. Monack: Stanford School of Medicine, Stanford University

Nature, 2012, vol. 490, issue 7419, 288-291

Abstract: Activation of the non-canonical, pro-inflammatory caspase-11 by Salmonella typhimurium is shown to contribute to bacterial spread and pathogenesis by the induction of macrophage cell death.

Date: 2012
References: Add references at CitEc
Citations:

Downloads: (external link)
https://www.nature.com/articles/nature11419 Abstract (text/html)
Access to the full text of the articles in this series is restricted.

Related works:
This item may be available elsewhere in EconPapers: Search for items with the same title.

Export reference: BibTeX RIS (EndNote, ProCite, RefMan) HTML/Text

Persistent link: https://EconPapers.repec.org/RePEc:nat:nature:v:490:y:2012:i:7419:d:10.1038_nature11419

Ordering information: This journal article can be ordered from
https://www.nature.com/

DOI: 10.1038/nature11419

Access Statistics for this article

Nature is currently edited by Magdalena Skipper

More articles in Nature from Nature
Bibliographic data for series maintained by Sonal Shukla () and Springer Nature Abstracting and Indexing ().