Mammalian heart renewal by pre-existing cardiomyocytes

Samuel E. Senyo, Matthew L. Steinhauser, Christie L. Pizzimenti, Vicky K. Yang, Lei Cai, Mei Wang, Ting-Di Wu, Jean-Luc Guerquin-Kern, Claude P. Lechene and Richard T. Lee ()
Additional contact information
Samuel E. Senyo: Brigham and Women’s Hospital and Harvard Medical School
Matthew L. Steinhauser: Brigham and Women’s Hospital and Harvard Medical School
Christie L. Pizzimenti: Brigham and Women’s Hospital and Harvard Medical School
Vicky K. Yang: Brigham and Women’s Hospital and Harvard Medical School
Lei Cai: Brigham and Women’s Hospital and Harvard Medical School
Mei Wang: National Resource for Imaging Mass Spectrometry
Ting-Di Wu: INSERM U.759
Jean-Luc Guerquin-Kern: INSERM U.759
Claude P. Lechene: National Resource for Imaging Mass Spectrometry
Richard T. Lee: Brigham and Women’s Hospital and Harvard Medical School

Nature, 2013, vol. 493, issue 7432, 433-436

Abstract: During normal ageing a low rate of division of pre-existing cardiomyocytes, rather than progenitor cells, is responsible for cardiomyocyte genesis; this process is increased fourfold during myocardial infarction.

Date: 2013
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DOI: 10.1038/nature11682

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