Landscape of the PARKIN-dependent ubiquitylome in response to mitochondrial depolarization

Shireen A. Sarraf, Malavika Raman, Virginia Guarani-Pereira, Mathew E. Sowa, Edward L. Huttlin, Steven P. Gygi and J. Wade Harper ()
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Shireen A. Sarraf: Harvard Medical School, 240 Longwood Avenue, Boston, Massachusetts 02115, USA
Malavika Raman: Harvard Medical School, 240 Longwood Avenue, Boston, Massachusetts 02115, USA
Virginia Guarani-Pereira: Harvard Medical School, 240 Longwood Avenue, Boston, Massachusetts 02115, USA
Mathew E. Sowa: Harvard Medical School, 240 Longwood Avenue, Boston, Massachusetts 02115, USA
Edward L. Huttlin: Harvard Medical School, 240 Longwood Avenue, Boston, Massachusetts 02115, USA
Steven P. Gygi: Harvard Medical School, 240 Longwood Avenue, Boston, Massachusetts 02115, USA
J. Wade Harper: Harvard Medical School, 240 Longwood Avenue, Boston, Massachusetts 02115, USA

Nature, 2013, vol. 496, issue 7445, 372-376

Abstract: PARKIN, a protein involved in mitochondria clearance by autophagy, is often mutated in early-onset familial Parkinson’s disease; here the cellular repertoire of PARKIN targets is identified by quantitative proteomics.

Date: 2013
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DOI: 10.1038/nature12043

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