Phosphatidylserine receptor BAI1 and apoptotic cells as new promoters of myoblast fusion

Amelia E. Hochreiter-Hufford, Chang Sup Lee, Jason M. Kinchen, Jennifer D. Sokolowski, Sanja Arandjelovic, Jarrod A. Call, Alexander L. Klibanov, Zhen Yan, James W. Mandell and Kodi S. Ravichandran ()
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Amelia E. Hochreiter-Hufford: Immunology, and Cancer Biology, University of Virginia, Charlottesville, Virginia 22908, USA
Chang Sup Lee: Immunology, and Cancer Biology, University of Virginia, Charlottesville, Virginia 22908, USA
Jason M. Kinchen: Immunology, and Cancer Biology, University of Virginia, Charlottesville, Virginia 22908, USA
Jennifer D. Sokolowski: Center for Cell Clearance, University of Virginia, Charlottesville, Virginia 22908, USA
Sanja Arandjelovic: Immunology, and Cancer Biology, University of Virginia, Charlottesville, Virginia 22908, USA
Jarrod A. Call: Robert M. Berne Cardiovascular Research Center, University of Virginia, Charlottesville, Virginia 22908, USA
Alexander L. Klibanov: Robert M. Berne Cardiovascular Research Center, University of Virginia, Charlottesville, Virginia 22908, USA
Zhen Yan: Robert M. Berne Cardiovascular Research Center, University of Virginia, Charlottesville, Virginia 22908, USA
James W. Mandell: Center for Cell Clearance, University of Virginia, Charlottesville, Virginia 22908, USA
Kodi S. Ravichandran: Immunology, and Cancer Biology, University of Virginia, Charlottesville, Virginia 22908, USA

Nature, 2013, vol. 497, issue 7448, 263-267

Abstract: The apoptotic event of phosphatidylserine exposure and its recognition by the receptor BAI1 has an unexpected new role as a signal enhancing mouse myoblast fusion, an insight with relevance to some congenital muscle diseases and muscle injury treatments.

Date: 2013
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DOI: 10.1038/nature12135

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