Microbiota-liberated host sugars facilitate post-antibiotic expansion of enteric pathogens

Katharine M. Ng, Jessica A. Ferreyra, Steven K. Higginbottom, Jonathan B. Lynch, Purna C. Kashyap, Smita Gopinath, Natasha Naidu, Biswa Choudhury, Bart C. Weimer, Denise M. Monack and Justin L. Sonnenburg ()
Additional contact information
Katharine M. Ng: Stanford University School of Medicine
Jessica A. Ferreyra: Stanford University School of Medicine
Steven K. Higginbottom: Stanford University School of Medicine
Jonathan B. Lynch: Stanford University School of Medicine
Purna C. Kashyap: Stanford University School of Medicine
Smita Gopinath: Stanford University School of Medicine
Natasha Naidu: Glycobiology Research and Training Center, University of California
Biswa Choudhury: Glycobiology Research and Training Center, University of California
Bart C. Weimer: University of California
Denise M. Monack: Stanford University School of Medicine
Justin L. Sonnenburg: Stanford University School of Medicine

Nature, 2013, vol. 502, issue 7469, 96-99

Abstract: Antibiotic treatment disturbs the commensal microbiota and is often followed by infection with enteric pathogens such as Salmonella typhimurium and Clostridium difficile; pathogen expansion is fuelled by antibiotic-driven accumulation of commensal-liberated host mucosal carbohydrates.

Date: 2013
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DOI: 10.1038/nature12503

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