Cell-cycle-regulated activation of Akt kinase by phosphorylation at its carboxyl terminus

Pengda Liu, Michael Begley, Wojciech Michowski, Hiroyuki Inuzuka, Miriam Ginzberg, Daming Gao, Peiling Tsou, Wenjian Gan, Antonella Papa, Byeong Mo Kim, Lixin Wan, Amrik Singh, Bo Zhai, Min Yuan, Zhiwei Wang, Steven P. Gygi, Tae Ho Lee, Kun-Ping Lu, Alex Toker, Pier Paolo Pandolfi, John M. Asara, Marc W. Kirschner, Piotr Sicinski, Lewis Cantley and Wenyi Wei ()
Additional contact information
Pengda Liu: Beth Israel Deaconess Medical Center, Harvard Medical School
Michael Begley: Beth Israel Deaconess Medical Center
Wojciech Michowski: Harvard Medical School
Hiroyuki Inuzuka: Beth Israel Deaconess Medical Center, Harvard Medical School
Miriam Ginzberg: Harvard Medical School
Daming Gao: Beth Israel Deaconess Medical Center, Harvard Medical School
Peiling Tsou: Beth Israel Deaconess Medical Center
Wenjian Gan: Beth Israel Deaconess Medical Center, Harvard Medical School
Antonella Papa: Beth Israel Deaconess Medical Center, Harvard Medical School
Byeong Mo Kim: Beth Israel Deaconess Medical Center
Lixin Wan: Beth Israel Deaconess Medical Center, Harvard Medical School
Amrik Singh: Cell Signaling Technology
Bo Zhai: Harvard Medical School
Min Yuan: Beth Israel Deaconess Medical Center
Zhiwei Wang: Beth Israel Deaconess Medical Center, Harvard Medical School
Steven P. Gygi: Harvard Medical School
Tae Ho Lee: Beth Israel Deaconess Medical Center
Kun-Ping Lu: Beth Israel Deaconess Medical Center
Alex Toker: Beth Israel Deaconess Medical Center, Harvard Medical School
Pier Paolo Pandolfi: Beth Israel Deaconess Medical Center, Harvard Medical School
John M. Asara: Beth Israel Deaconess Medical Center
Marc W. Kirschner: Harvard Medical School
Piotr Sicinski: Harvard Medical School
Lewis Cantley: Beth Israel Deaconess Medical Center
Wenyi Wei: Beth Israel Deaconess Medical Center, Harvard Medical School

Nature, 2014, vol. 508, issue 7497, 541-545

Abstract: Phosphorylation of Akt at its carboxy-terminal tail is an essential layer of Akt activation to regulate its physiological functions.

Date: 2014
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DOI: 10.1038/nature13079

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