CFIm25 links alternative polyadenylation to glioblastoma tumour suppression

Chioniso P. Masamha, Zheng Xia, Jingxuan Yang, Todd R. Albrecht, Min Li, Ann-Bin Shyu (), Wei Li () and Eric J. Wagner ()
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Chioniso P. Masamha: The University of Texas Medical School at Houston
Zheng Xia: Baylor College of Medicine
Jingxuan Yang: The University of Texas Medical School at Houston
Todd R. Albrecht: The University of Texas Medical School at Houston
Min Li: The University of Texas Medical School at Houston
Ann-Bin Shyu: The University of Texas Medical School at Houston
Wei Li: Baylor College of Medicine
Eric J. Wagner: The University of Texas Medical School at Houston

Nature, 2014, vol. 510, issue 7505, 412-416

Abstract: CFIm25 is identified as a factor that prevents messenger RNAs being shortened due to altered 3′ polyadenylation, which typically occurs when cells undergo high proliferation and correlates with increased tumorigenic activity in glioblastoma tumours.

Date: 2014
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DOI: 10.1038/nature13261

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