A long noncoding RNA protects the heart from pathological hypertrophy

Pei Han, Wei Li, Chiou-Hong Lin, Jin Yang, Ching Shang, Sylvia T. Nurnberg, Kevin Kai Jin, Weihong Xu, Chieh-Yu Lin, Chien-Jung Lin, Yiqin Xiong, Huan-Chieh Chien, Bin Zhou, Euan Ashley, Daniel Bernstein, Peng-Sheng Chen, Huei-Sheng Vincent Chen, Thomas Quertermous and Ching-Pin Chang ()
Additional contact information
Pei Han: Indiana University School of Medicine
Wei Li: Indiana University School of Medicine
Chiou-Hong Lin: Cardiovascular Institute, Stanford University School of Medicine
Jin Yang: Indiana University School of Medicine
Ching Shang: Cardiovascular Institute, Stanford University School of Medicine
Sylvia T. Nurnberg: Cardiovascular Institute, Stanford University School of Medicine
Kevin Kai Jin: Cardiovascular Institute, Stanford University School of Medicine
Weihong Xu: Stanford Genome Technology Center, Stanford University School of Medicine
Chieh-Yu Lin: Cardiovascular Institute, Stanford University School of Medicine
Chien-Jung Lin: Cardiovascular Institute, Stanford University School of Medicine
Yiqin Xiong: Cardiovascular Institute, Stanford University School of Medicine
Huan-Chieh Chien: Cardiovascular Institute, Stanford University School of Medicine
Bin Zhou: Pediatrics, and Medicine (Cardiology), Albert Einstein College of Medicine of Yeshiva University, 1301 Morris Park Avenue, Price Center 420
Euan Ashley: Cardiovascular Institute, Stanford University School of Medicine
Daniel Bernstein: Stanford University School of Medicine
Peng-Sheng Chen: Indiana University School of Medicine
Huei-Sheng Vincent Chen: Del E. Webb Neuroscience, Aging & Stem Cell Research Center, Sanford/Burnham Medical Research Institute
Thomas Quertermous: Cardiovascular Institute, Stanford University School of Medicine
Ching-Pin Chang: Indiana University School of Medicine

Nature, 2014, vol. 514, issue 7520, 102-106

Abstract: Here, a long noncoding RNA, termed Mhrt, is identified in the loci of myosin heavy chain (Myh) genes in mice and shown to be capable of suppressing cardiomyopathy in the animals, as well as being repressed in diseased human hearts.

Date: 2014
References: Add references at CitEc
Citations: View citations in EconPapers (1)

Downloads: (external link)
https://www.nature.com/articles/nature13596 Abstract (text/html)
Access to the full text of the articles in this series is restricted.

Related works:
This item may be available elsewhere in EconPapers: Search for items with the same title.

Export reference: BibTeX RIS (EndNote, ProCite, RefMan) HTML/Text

Persistent link: https://EconPapers.repec.org/RePEc:nat:nature:v:514:y:2014:i:7520:d:10.1038_nature13596

Ordering information: This journal article can be ordered from
https://www.nature.com/

DOI: 10.1038/nature13596

Access Statistics for this article

Nature is currently edited by Magdalena Skipper

More articles in Nature from Nature
Bibliographic data for series maintained by Sonal Shukla () and Springer Nature Abstracting and Indexing ().