Tuft-cell-derived IL-25 regulates an intestinal ILC2–epithelial response circuit

Jakob von Moltke, Ming Ji, Hong-Erh Liang and Richard M. Locksley ()
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Jakob von Moltke: University of California San Francisco
Ming Ji: University of California San Francisco
Hong-Erh Liang: University of California San Francisco
Richard M. Locksley: University of California San Francisco

Nature, 2016, vol. 529, issue 7585, 221-225

Abstract: Epithelial tuft cells are shown to be the source of intestinal interleukin (IL)-25 that is required for activation of type 2 innate lymphoid cells (ILC2s), ILC2-regulated tuft and goblet cell expansion, and control of parasite infection.

Date: 2016
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DOI: 10.1038/nature16161

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