A phosphoinositide conversion mechanism for exit from endosomes

Katharina Ketel, Michael Krauss, Anne-Sophie Nicot, Dmytro Puchkov, Marnix Wieffer, Rainer Müller, Devaraj Subramanian, Carsten Schultz, Jocelyn Laporte and Volker Haucke ()
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Katharina Ketel: Leibniz-Institut für Molekulare Pharmakologie
Michael Krauss: Leibniz-Institut für Molekulare Pharmakologie
Anne-Sophie Nicot: Institut de Genetique et de Biologie Moleculaire et Cellulaire (IGBMC), INSERM U964, CNRS UMR7104, Strasbourg University
Dmytro Puchkov: Leibniz-Institut für Molekulare Pharmakologie
Marnix Wieffer: Freie Universität Berlin, Faculty of Biology, Chemistry and Pharmacy
Rainer Müller: European Molecular Biology Laboratory (EMBL), Cell Biology and Biophysics Unit
Devaraj Subramanian: European Molecular Biology Laboratory (EMBL), Cell Biology and Biophysics Unit
Carsten Schultz: European Molecular Biology Laboratory (EMBL), Cell Biology and Biophysics Unit
Jocelyn Laporte: Institut de Genetique et de Biologie Moleculaire et Cellulaire (IGBMC), INSERM U964, CNRS UMR7104, Strasbourg University
Volker Haucke: Leibniz-Institut für Molekulare Pharmakologie

Nature, 2016, vol. 529, issue 7586, 408-412

Abstract: A mechanism for phosphoinositide conversion at endosomes to enable exit from the endosomal system, suggesting that defective phosphoinositide conversion at endosomes underlies X-linked centronuclear myopathy.

Date: 2016
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DOI: 10.1038/nature16516

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