Correction: Corrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells

Stephanie C. Eisenbarth, Adam Williams, Oscar R. Colegio, Hailong Meng, Till Strowig, Anthony Rongvaux, Jorge Henao-Mejia, Christoph A. Thaiss, Sophie Joly, David G. Gonzalez, Lan Xu, Lauren A. Zenewicz, Ann M. Haberman, Eran Elinav, Steven H. Kleinstein, Fayyaz S. Sutterwala and Richard A. Flavell

Nature, 2016, vol. 530, issue 7591, 504-504

Abstract: Nature 484, 510–513 (2012); 10.1038/nature11012 In this Letter, we reported that NLRP10-deficient mice had no defect in inflammasome function in macrophages or dendritic cells (DCs). Instead, a loss of T-cell-dependent immune responses was seen in these mice secondary to a defect in DC migration. Wehave since noticed a change in the phenotype of the NLRP10-knockout mice involving DC migration, after backcrossing them onto backgrounds such as FVB or BALB/c (see Supplementary Methods).

Date: 2016
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DOI: 10.1038/nature16074

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