Hepatitis B virus X protein identifies the Smc5/6 complex as a host restriction factor

Adrien Decorsière, Henrik Mueller, Pieter C. van Breugel, Fabien Abdul, Laetitia Gerossier, Rudolf K. Beran, Christine M. Livingston, Congrong Niu, Simon P. Fletcher (), Olivier Hantz () and Michel Strubin ()
Additional contact information
Adrien Decorsière: University Medical Centre (C.M.U.)
Henrik Mueller: University Medical Centre (C.M.U.)
Pieter C. van Breugel: University Medical Centre (C.M.U.)
Fabien Abdul: University Medical Centre (C.M.U.)
Laetitia Gerossier: CRCL, INSERM U1052, CNRS 5286, Université de Lyon
Rudolf K. Beran: Gilead Sciences, Inc.
Christine M. Livingston: Gilead Sciences, Inc.
Congrong Niu: Gilead Sciences, Inc.
Simon P. Fletcher: Gilead Sciences, Inc.
Olivier Hantz: CRCL, INSERM U1052, CNRS 5286, Université de Lyon
Michel Strubin: University Medical Centre (C.M.U.)

Nature, 2016, vol. 531, issue 7594, 386-389

Abstract: Hepatitis B virus X protein stimulates transcription from the viral DNA episome by hijacking the host ubiquitin machinery to target the Smc5/6 complex for degradation.

Date: 2016
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DOI: 10.1038/nature17170

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