Mitochondrial ROS regulate thermogenic energy expenditure and sulfenylation of UCP1

Edward T. Chouchani, Lawrence Kazak, Mark P. Jedrychowski, Gina Z. Lu, Brian K. Erickson, John Szpyt, Kerry A. Pierce, Dina Laznik-Bogoslavski, Ramalingam Vetrivelan, Clary B. Clish, Alan J. Robinson, Steve P. Gygi and Bruce M. Spiegelman ()
Additional contact information
Edward T. Chouchani: Dana-Farber Cancer Institute, Harvard Medical School
Lawrence Kazak: Dana-Farber Cancer Institute, Harvard Medical School
Mark P. Jedrychowski: Harvard Medical School
Gina Z. Lu: Dana-Farber Cancer Institute, Harvard Medical School
Brian K. Erickson: Harvard Medical School
John Szpyt: Harvard Medical School
Kerry A. Pierce: Broad Institute of Harvard and MIT
Dina Laznik-Bogoslavski: Dana-Farber Cancer Institute, Harvard Medical School
Ramalingam Vetrivelan: Harvard Medical School
Clary B. Clish: Broad Institute of Harvard and MIT
Alan J. Robinson: MRC Mitochondrial Biology Unit
Steve P. Gygi: Harvard Medical School
Bruce M. Spiegelman: Dana-Farber Cancer Institute, Harvard Medical School

Nature, 2016, vol. 532, issue 7597, 112-116

Abstract: Uncoupling protein 1 (UCP1)-dependent thermogenesis in brown adipose tissue is supported by a burst of mitochondrial reactive oxygen species upon cold exposure.

Date: 2016
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DOI: 10.1038/nature17399

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