TAM receptors regulate multiple features of microglial physiology

Lawrence Fourgeaud, Paqui G. Través, Yusuf Tufail, Humberto Leal-Bailey, Erin D. Lew, Patrick G. Burrola, Perri Callaway, Anna Zagórska, Carla V. Rothlin (), Axel Nimmerjahn and Greg Lemke ()
Additional contact information
Lawrence Fourgeaud: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies
Paqui G. Través: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies
Yusuf Tufail: Waitt Advanced Biophotonics Center, The Salk Institute for Biological Studies
Humberto Leal-Bailey: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies
Erin D. Lew: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies
Patrick G. Burrola: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies
Perri Callaway: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies
Anna Zagórska: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies
Carla V. Rothlin: Yale University School of Medicine
Axel Nimmerjahn: Waitt Advanced Biophotonics Center, The Salk Institute for Biological Studies
Greg Lemke: Molecular Neurobiology Laboratory, The Salk Institute for Biological Studies

Nature, 2016, vol. 532, issue 7598, 240-244

Abstract: Microglial phagocytosis is required for neurogenic niche maintenance and response to injury; the TAM kinases Mer and Axl are expressed by microglia in the adult CNS, and mediate the clearance of apoptotic cells from the niche.

Date: 2016
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DOI: 10.1038/nature17630

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