NOD1 and NOD2 signalling links ER stress with inflammation

A. Marijke Keestra-Gounder, Mariana X. Byndloss, Núbia Seyffert, Briana M. Young, Alfredo Chávez-Arroyo, April Y. Tsai, Stephanie A. Cevallos, Maria G. Winter, Oanh H. Pham, Connor R. Tiffany, Maarten F. de Jong, Tobias Kerrinnes, Resmi Ravindran, Paul A. Luciw, Stephen J. McSorley, Andreas J. Bäumler and Renée M. Tsolis ()
Additional contact information
A. Marijke Keestra-Gounder: School of Medicine, University of California at Davis
Mariana X. Byndloss: School of Medicine, University of California at Davis
Núbia Seyffert: School of Medicine, University of California at Davis
Briana M. Young: School of Medicine, University of California at Davis
Alfredo Chávez-Arroyo: School of Medicine, University of California at Davis
April Y. Tsai: School of Medicine, University of California at Davis
Stephanie A. Cevallos: School of Medicine, University of California at Davis
Maria G. Winter: School of Medicine, University of California at Davis
Oanh H. Pham: Center for Comparative Medicine, Schools of Medicine and Veterinary Medicine, University of California at Davis
Connor R. Tiffany: School of Medicine, University of California at Davis
Maarten F. de Jong: School of Medicine, University of California at Davis
Tobias Kerrinnes: School of Medicine, University of California at Davis
Resmi Ravindran: Center for Comparative Medicine, Schools of Medicine and Veterinary Medicine, University of California at Davis
Paul A. Luciw: Center for Comparative Medicine, Schools of Medicine and Veterinary Medicine, University of California at Davis
Stephen J. McSorley: Center for Comparative Medicine, Schools of Medicine and Veterinary Medicine, University of California at Davis
Andreas J. Bäumler: School of Medicine, University of California at Davis
Renée M. Tsolis: School of Medicine, University of California at Davis

Nature, 2016, vol. 532, issue 7599, 394-397

Abstract: A novel link between the unfolded protein response and NOD1/2 innate immune signalling, showing that NOD1/2 are required for ER-stress-induced IL-6 production in response to infection with Brucella abortus.

Date: 2016
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DOI: 10.1038/nature17631

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