Tumour-cell-induced endothelial cell necroptosis via death receptor 6 promotes metastasis

Boris Strilic (), Lida Yang, Julián Albarrán-Juárez, Laurens Wachsmuth, Kang Han, Ulrike C. Müller, Manolis Pasparakis and Stefan Offermanns ()
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Boris Strilic: Max Planck Institute for Heart and Lung Research
Lida Yang: Max Planck Institute for Heart and Lung Research
Julián Albarrán-Juárez: Max Planck Institute for Heart and Lung Research
Laurens Wachsmuth: University of Cologne, Institute for Genetics, Center for Molecular Medicine (CMMC), and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), Joseph-Stelzmann-Strasse 26, 50931 Cologne, Germany
Kang Han: University of Heidelberg, Institute for Pharmacy and Molecular Biotechnology, Im Neuenheimer Feld 364
Ulrike C. Müller: University of Heidelberg, Institute for Pharmacy and Molecular Biotechnology, Im Neuenheimer Feld 364
Manolis Pasparakis: University of Cologne, Institute for Genetics, Center for Molecular Medicine (CMMC), and Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), Joseph-Stelzmann-Strasse 26, 50931 Cologne, Germany
Stefan Offermanns: Max Planck Institute for Heart and Lung Research

Nature, 2016, vol. 536, issue 7615, 215-218

Abstract: Human and murine tumour cells induce programmed necrosis (necroptosis) of endothelial cells, which promotes tumour cell extravasation and metastasis.

Date: 2016
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DOI: 10.1038/nature19076

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