Rho GTPase complementation underlies BDNF-dependent homo- and heterosynaptic plasticity

Nathan G. Hedrick, Stephen C. Harward, Charles E. Hall, Hideji Murakoshi, James O. McNamara and Ryohei Yasuda ()
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Nathan G. Hedrick: Duke University Medical Center, Research Drive
Stephen C. Harward: Duke University Medical Center, Research Drive
Charles E. Hall: Duke University Medical Center, Research Drive
Hideji Murakoshi: National Institute for Physiological Science, Myodaiji
James O. McNamara: Duke University Medical Center, Research Drive
Ryohei Yasuda: Duke University Medical Center, Research Drive

Nature, 2016, vol. 538, issue 7623, 104-108

Abstract: The three small GTPases Rac1, RhoA and Cdc42 are differentially involved in structural long-term potentiation of rodent dendritic spines, simultaneously ensuring signal specificity and also priming the system for plasticity.

Date: 2016
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DOI: 10.1038/nature19784

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