Control of mitochondrial function and cell growth by the atypical cadherin Fat1

Longyue L. Cao, Dario F. Riascos-Bernal, Prameladevi Chinnasamy, Charlene M. Dunaway, Rong Hou, Mario A. Pujato, Brian P. O’Rourke, Veronika Miskolci, Liang Guo, Louis Hodgson, Andras Fiser and Nicholas E. S. Sibinga ()
Additional contact information
Longyue L. Cao: Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine
Dario F. Riascos-Bernal: Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine
Prameladevi Chinnasamy: Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine
Charlene M. Dunaway: Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine
Rong Hou: Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine
Mario A. Pujato: Albert Einstein College of Medicine
Brian P. O’Rourke: Albert Einstein College of Medicine
Veronika Miskolci: Albert Einstein College of Medicine
Liang Guo: CVPath Institute
Louis Hodgson: Albert Einstein College of Medicine
Andras Fiser: Albert Einstein College of Medicine
Nicholas E. S. Sibinga: Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine

Nature, 2016, vol. 539, issue 7630, 575-578

Abstract: Fragments of the atypical cadherin Fat1 accumulate in the mitochondria of vascular smooth muscle cells where they reduce respiration, leading to a regulated proliferative response to arterial injury.

Date: 2016
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DOI: 10.1038/nature20170

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