EPRS is a critical mTORC1–S6K1 effector that influences adiposity in mice

Abul Arif, Fulvia Terenzi, Alka A. Potdar, Jie Jia, Jessica Sacks, Arnab China, Dalia Halawani, Kommireddy Vasu, Xiaoxia Li, J. Mark Brown, Jie Chen, Sara C. Kozma, George Thomas and Paul L. Fox ()
Additional contact information
Abul Arif: Lerner Research Institute, Cleveland Clinic
Fulvia Terenzi: Lerner Research Institute, Cleveland Clinic
Alka A. Potdar: F. Widjaja Foundation Inflammatory Bowel and Immunobiology Research Institute, Cedars-Sinai Medical Center
Jie Jia: Lerner Research Institute, Cleveland Clinic
Jessica Sacks: Lerner Research Institute, Cleveland Clinic
Arnab China: Lerner Research Institute, Cleveland Clinic
Dalia Halawani: Lerner Research Institute, Cleveland Clinic
Kommireddy Vasu: Lerner Research Institute, Cleveland Clinic
Xiaoxia Li: Lerner Research Institute, Cleveland Clinic
J. Mark Brown: Lerner Research Institute, Cleveland Clinic
Jie Chen: University of Illinois
Sara C. Kozma: Catalan Institute of Oncology, ICO, Bellvitge Biomedical Research Institute, IDIBELL
George Thomas: Catalan Institute of Oncology, ICO, Bellvitge Biomedical Research Institute, IDIBELL
Paul L. Fox: Lerner Research Institute, Cleveland Clinic

Nature, 2017, vol. 542, issue 7641, 357-361

Abstract: Glutamyl-prolyl tRNA synthetase (EPRS) is a downstream effector of the mTORC1–S6K1 signalling axis and contributes to adiposity and ageing in mice.

Date: 2017
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DOI: 10.1038/nature21380

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