DHX9 suppresses RNA processing defects originating from the Alu invasion of the human genome

Tuğçe Aktaş, İbrahim Avşar Ilık, Daniel Maticzka, Vivek Bhardwaj, Cecilia Pessoa Rodrigues, Gerhard Mittler, Thomas Manke, Rolf Backofen and Asifa Akhtar ()
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Tuğçe Aktaş: Max Planck Institute of Immunobiology and Epigenetics
İbrahim Avşar Ilık: Max Planck Institute of Immunobiology and Epigenetics
Daniel Maticzka: Institute for Informatics, Albert-Ludwigs-University
Vivek Bhardwaj: Max Planck Institute of Immunobiology and Epigenetics
Cecilia Pessoa Rodrigues: Max Planck Institute of Immunobiology and Epigenetics
Gerhard Mittler: Max Planck Institute of Immunobiology and Epigenetics
Thomas Manke: Max Planck Institute of Immunobiology and Epigenetics
Rolf Backofen: Institute for Informatics, Albert-Ludwigs-University
Asifa Akhtar: Max Planck Institute of Immunobiology and Epigenetics

Nature, 2017, vol. 544, issue 7648, 115-119

Abstract: In the absence of DHX9, circular RNAs accumulate and transcription and translation are dysregulated—effects that are exacerbated by concomitant depletion of the RNA-editing enzyme ADAR.

Date: 2017
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DOI: 10.1038/nature21715

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