High-avidity IgA protects the intestine by enchaining growing bacteria

Kathrin Moor, Médéric Diard, Mikael E. Sellin, Boas Felmy, Sandra Y. Wotzka, Albulena Toska, Erik Bakkeren, Markus Arnoldini, Florence Bansept, Alma Dal Co, Tom Völler, Andrea Minola, Blanca Fernandez-Rodriguez, Gloria Agatic, Sonia Barbieri, Luca Piccoli, Costanza Casiraghi, Davide Corti, Antonio Lanzavecchia, Roland R. Regoes, Claude Loverdo, Roman Stocker, Douglas R. Brumley, Wolf-Dietrich Hardt and Emma Slack
Additional contact information
Kathrin Moor: Institute of Microbiology, ETH Zürich
Médéric Diard: Institute of Microbiology, ETH Zürich
Mikael E. Sellin: Institute of Microbiology, ETH Zürich
Boas Felmy: Institute of Microbiology, ETH Zürich
Sandra Y. Wotzka: Institute of Microbiology, ETH Zürich
Albulena Toska: Institute of Microbiology, ETH Zürich
Erik Bakkeren: Institute of Microbiology, ETH Zürich
Markus Arnoldini: Institute of Microbiology, ETH Zürich
Florence Bansept: Laboratoire Jean Perrin (UMR 8237), CNRS - UPMC
Alma Dal Co: ETH Zurich
Tom Völler: Institute of Microbiology, ETH Zürich
Andrea Minola: Humabs BioMed SA
Blanca Fernandez-Rodriguez: Institute for Research in Biomedicine
Gloria Agatic: Humabs BioMed SA
Sonia Barbieri: Institute for Research in Biomedicine
Luca Piccoli: Institute for Research in Biomedicine
Costanza Casiraghi: Institute for Research in Biomedicine
Davide Corti: Humabs BioMed SA
Antonio Lanzavecchia: Institute of Microbiology, ETH Zürich
Roland R. Regoes: Institute of Integrative Biology, ETH Zürich
Claude Loverdo: Laboratoire Jean Perrin (UMR 8237), CNRS - UPMC
Roman Stocker: Institute of Environmental Engineering, Environmental, and Geomatic Engineering, ETH Zürich
Douglas R. Brumley: Institute of Environmental Engineering, Environmental, and Geomatic Engineering, ETH Zürich
Wolf-Dietrich Hardt: Institute of Microbiology, ETH Zürich
Emma Slack: Institute of Microbiology, ETH Zürich

Nature, 2017, vol. 544, issue 7651, 498-502

Abstract: Oral-vaccine-induced IgA cross-links growing bacteria into clonal aggregates, inhibiting pathogenesis, adaption and the spread of antimicrobial resistance genes.

Date: 2017
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DOI: 10.1038/nature22058

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