Maternal gut bacteria promote neurodevelopmental abnormalities in mouse offspring

Sangdoo Kim, Hyunju Kim, Yeong Shin Yim, Soyoung Ha, Koji Atarashi, Tze Guan Tan, Randy S. Longman, Kenya Honda, Dan R. Littman, Gloria B. Choi () and Jun R. Huh ()
Additional contact information
Sangdoo Kim: University of Massachusetts Medical School
Hyunju Kim: University of Massachusetts Medical School
Yeong Shin Yim: The McGovern Institute for Brain Research, Massachusetts Institute of Technology
Soyoung Ha: University of Massachusetts Medical School
Koji Atarashi: Keio University School of Medicine
Tze Guan Tan: Harvard Medical School
Randy S. Longman: The Jill Roberts Institute for Research in IBD, Weill Cornell Medicine
Kenya Honda: Keio University School of Medicine
Dan R. Littman: The Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine
Gloria B. Choi: The McGovern Institute for Brain Research, Massachusetts Institute of Technology
Jun R. Huh: University of Massachusetts Medical School

Nature, 2017, vol. 549, issue 7673, 528-532

Abstract: Maternal immune activation (MIA)-mediated abnormal behavioural phenotypes require defined gut commensal bacteria for the induction of IL-17-producing T helper 17 cells.

Date: 2017
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DOI: 10.1038/nature23910

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