Regulation of DNA repair pathway choice in S and G2 phases by the NHEJ inhibitor CYREN
Nausica Arnoult,
Adriana Correia,
Jiao Ma,
Anna Merlo,
Sara Garcia-Gomez,
Marija Maric,
Marco Tognetti,
Christopher W. Benner,
Simon J. Boulton,
Alan Saghatelian and
Jan Karlseder ()
Additional contact information
Nausica Arnoult: The Salk Institute for Biological Studies
Adriana Correia: The Salk Institute for Biological Studies
Jiao Ma: The Salk Institute for Biological Studies
Anna Merlo: The Salk Institute for Biological Studies
Sara Garcia-Gomez: Dsb Repair Metabolism Laboratory, The Francis Crick Institute
Marija Maric: Dsb Repair Metabolism Laboratory, The Francis Crick Institute
Marco Tognetti: The Salk Institute for Biological Studies
Christopher W. Benner: University of California San Diego
Simon J. Boulton: Dsb Repair Metabolism Laboratory, The Francis Crick Institute
Alan Saghatelian: The Salk Institute for Biological Studies
Jan Karlseder: The Salk Institute for Biological Studies
Nature, 2017, vol. 549, issue 7673, 548-552
Abstract:
CYREN is a direct inhibitor of classical non-homologous end joining that promotes error-free repair by homologous recombination during the S and G2 phases of the cell cycle.
Date: 2017
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Persistent link: https://EconPapers.repec.org/RePEc:nat:nature:v:549:y:2017:i:7673:d:10.1038_nature24023
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DOI: 10.1038/nature24023
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