Immune evasion of Plasmodium falciparum by RIFIN via inhibitory receptors

Fumiji Saito, Kouyuki Hirayasu, Takeshi Satoh, Christian W. Wang, John Lusingu, Takao Arimori, Kyoko Shida, Nirianne Marie Q. Palacpac, Sawako Itagaki, Shiroh Iwanaga, Eizo Takashima, Takafumi Tsuboi, Masako Kohyama, Tadahiro Suenaga, Marco Colonna, Junichi Takagi, Thomas Lavstsen, Toshihiro Horii and Hisashi Arase ()
Additional contact information
Fumiji Saito: Research Institute for Microbial Diseases, Osaka University, Suita
Kouyuki Hirayasu: Laboratory of Immunochemistry, WPI Immunology Frontier Research Centre, Osaka University, Suita
Takeshi Satoh: Research Institute for Microbial Diseases, Osaka University, Suita
Christian W. Wang: Centre for Medical Parasitology, University of Copenhagen
John Lusingu: National Institute for Medical Research, Tanga Research Centre
Takao Arimori: Laboratory of Protein Synthesis and Expression, Institute for Protein Research, Osaka University
Kyoko Shida: Laboratory of Immunochemistry, WPI Immunology Frontier Research Centre, Osaka University, Suita
Nirianne Marie Q. Palacpac: Research Institute for Microbial Diseases, Osaka University
Sawako Itagaki: Research Institute for Microbial Diseases, Osaka University
Shiroh Iwanaga: School of Medicine, Mie University
Eizo Takashima: Proteo-Science Centre, Ehime University
Takafumi Tsuboi: Proteo-Science Centre, Ehime University
Masako Kohyama: Research Institute for Microbial Diseases, Osaka University, Suita
Tadahiro Suenaga: Research Institute for Microbial Diseases, Osaka University, Suita
Marco Colonna: Washington University School of Medicine
Junichi Takagi: Laboratory of Protein Synthesis and Expression, Institute for Protein Research, Osaka University
Thomas Lavstsen: Centre for Medical Parasitology, University of Copenhagen
Toshihiro Horii: Research Institute for Microbial Diseases, Osaka University
Hisashi Arase: Research Institute for Microbial Diseases, Osaka University, Suita

Nature, 2017, vol. 552, issue 7683, 101-105

Abstract: Proteins expressed on the surfaces of erythrocytes infected with Plasmodium falciparum help the parasite to evade the host immune system by acting as ligands for immune inhibitory receptors and thereby downregulating the immune response.

Date: 2017
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DOI: 10.1038/nature24994

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