The role of CD32 during HIV-1 infection

Lynn N. Bertagnolli, Jennifer A. White, Francesco R. Simonetti, Subul A. Beg, Jun Lai, Costin Tomescu, Alexandra J. Murray, Annukka A. R. Antar, Hao Zhang, Joseph B. Margolick, Rebecca Hoh, Stephen G. Deeks, Pablo Tebas, Luis J. Montaner, Robert F. Siliciano (), Gregory M. Laird and Janet D. Siliciano
Additional contact information
Lynn N. Bertagnolli: Johns Hopkins University School of Medicine
Jennifer A. White: Johns Hopkins University School of Medicine
Francesco R. Simonetti: Johns Hopkins University School of Medicine
Subul A. Beg: Johns Hopkins University School of Medicine
Jun Lai: Johns Hopkins University School of Medicine
Costin Tomescu: The Wistar Institute
Alexandra J. Murray: Johns Hopkins University School of Medicine
Annukka A. R. Antar: Johns Hopkins University School of Medicine
Hao Zhang: Johns Hopkins Bloomberg School of Public Health
Joseph B. Margolick: Johns Hopkins Bloomberg School of Public Health
Rebecca Hoh: University of California
Stephen G. Deeks: University of California
Pablo Tebas: Perelman School of Medicine at the University of Pennsylvania
Luis J. Montaner: The Wistar Institute
Robert F. Siliciano: Johns Hopkins University School of Medicine
Gregory M. Laird: Johns Hopkins University School of Medicine
Janet D. Siliciano: Johns Hopkins University School of Medicine

Nature, 2018, vol. 561, issue 7723, E17-E19

Keywords: Quantitative Viral Outgrowth Assay; CD25hi Cells; Negative Depletion; Replication-competent Provirus; Standard ELISA (search for similar items in EconPapers)
Date: 2018
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DOI: 10.1038/s41586-018-0494-3

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